Cardiology Conference-CVC 2026

Obstructive sleep apnea (OSA) is surprisingly prevalent and is defined by repeated episodes of upper airway blockage during sleep. These interruptions fragment sleep, reduce oxygen levels, and activate the body’s sympathetic nervous system. The consequences? A considerably higher risk of hypertension, arrhythmias, heart failure, coronary artery disease, and stroke. The ongoing cycles of low oxygen and disrupted sleep provoke inflammation, oxidative stress, and problems with blood vessels—each one nudging cardiovascular disease along its path. Early identification and intervention are crucial for reducing cardiovascular risk and improving patient outcomes.

Management of sleep apnea typically incorporates lifestyle adjustments, such as weight loss, regular exercise, and avoidance of alcohol or sedatives before sleep. Continuous positive airway pressure (CPAP) therapy remains the standard, while oral appliances and, in select cases, surgical procedures may be considered. Rigorous monitoring and management of comorbid conditions like hypertension, diabetes, and dyslipidemia are also essential. A collaborative, multidisciplinary approach—engaging cardiologists, pulmonologists, and sleep specialists—optimizes care. By addressing both sleep apnea and its cardiovascular consequences, clinicians can improve sleep quality, reduce the burden of heart disease, and enhance long-term cardiovascular health.