Cardiology Conference-CVC 2026

Air pollution stands out as a substantial and often underestimated contributor to cardiovascular disease. It is linked to increased incidences of heart attacks, strokes, hypertension, arrhythmias, and atherosclerosis. Major pollutants—such as fine particulate matter (PM2.5) and nitrogen dioxide—provoke inflammation, oxidative stress, and endothelial dysfunction. These processes collectively disrupt cardiovascular homeostasis and compromise vascular health.

Prolonged exposure to polluted air is associated with a higher risk of chronic cardiovascular conditions, while acute spikes in pollution have been implicated in triggering sudden cardiac events. Individuals residing in urban environments or industrial zones are disproportionately affected, with heightened vulnerability observed in older adults, those with pre-existing cardiovascular disease, and individuals diagnosed with metabolic syndrome.

Addressing this public health challenge requires both individual and systemic interventions. On a personal level, minimizing outdoor activities during periods of high pollution, utilizing air purification devices, and adopting lifestyle measures supportive of cardiovascular health—such as regular physical activity and balanced nutrition—are recommended. On a societal scale, policy initiatives aimed at reducing emissions, investing in sustainable energy sources, and improving overall air quality are essential.

Healthcare professionals play an important role by incorporating environmental risk assessment into cardiovascular care. Patient education about the cardiovascular effects of air pollution and the importance of preventive strategies can help mitigate risk. By integrating environmental factors with more traditional cardiovascular risk management, clinicians can contribute to reducing the global burden of cardiovascular disease and promote sustained heart health across vulnerable populations.